Search results for " MMP2"

showing 5 items of 5 documents

Another function of Hsp70 in mesoangioblast stem cells

2010

In recent years, it has been demonstrated that Hsp70 is released in the extracellular space under normal cell culture conditions, and this release is mediated through exosomes. We have demonstrated that A6 cells, a clone of mouse mesoangioblasts, produce and release in the extracellular space membrane vesicles, independently of culture growth conditions. These vesicles contains both structural proteins and biological active molecules, such as FGF-2 and the metalloproteinases MMP 2 and 9. We have also demonstrated that A6 vesicles contain HspP70 and its release is highly regulated. Some of the intracellular Hsp70 is localized on lipid rafts and its concentration in insoluble fraction increas…

Hsp70 MMP2/9
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Hsp70 level regulates MMP2 expression in mesoangioblast stemj cells

2013

Hsp70 mesoangioblasts stem cells MMP2
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MMP2 expression in mouse mesoangioblast is dependent on Hsp70 level.

2013

Mesoangioblast MMP2 Hsp70
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Abstracts of the 3rd Joint Meeting of Pathology and Laboratory Medicine

2016

Background: Degenerative forms of mitral valve diseases (MVDs) are complex pathologies. Thus, it is difficult to make generalizations about MVD pathways or genetic risk factors. However, a key role of metalloproteinases (MMPs) in their pathophysiology is emerging. Thus, we performed a study to assess eventual associations of some functional SNPs in MMP-2 and MMP-9 genes with MVD risk, symptom severity and short- and long-term (4.8 years) complications. Methods: For this purpose, 90 patients and two control groups were genotyped for MMP-2 and MMP-9 gene SNPs, and systemic levels of proatrial natriuretic peptide (ANP), and two enzymes were quantified and correlated to the MMP-2 and MMP-9 SNPs…

Pathologymedicine.medical_specialtybusiness.industryMedical laboratorySettore MED/05 - Patologia ClinicaMedicineJoint (building)businessDegenerative Mitral Valve Disease MMP2 and 9 polymorphismsPathology and Forensic MedicineThe American Journal of Pathology
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AGE6. Skin Ageing: Focus on the Role of Inflammatory Genetic Factors in Cutaneous Neoplasia

2014

AGE6. Skin Ageing: Focus on the Role of Inflammatory Genetic Factors in Cutaneous Neoplasia C. M. Gambino1, F. Crapanzano1, G. Accardi1, A. Aiello1, C. Virruso1, G. Pistone1, M. R. Bongiorno1, D. Lio1, C. R. Balistreri1, G. Candore1 1University of Palermo, Palermo, Italy Background: Skin aging is a complex process that involves intrinsic and exogenous causes. Photo-oxidative damage caused by UV is the leading cause of extrinsic aging of the skin, known as photo-ageing. UV damages can be linked mostly to overproduction of ROS that induces a complex molecular cascade able to accelerate physiological aging, determining a typical dermal/epidermal inflammation with an increased risk of getting s…

rs.4986790) TLR4 (+1196 C/T rs.4986791) MMP2 (-1306 C/T rs.243865) and MMP9 (-1562 C/T rs.3918242)SNPsSkin aging TLR4 (+896 A/G; rs.4986790) TLR4 (+1196 C/T rs.4986791) MMP2 (-1306 C/T rs.243865) and MMP9 (-1562 C/T rs.3918242)SNPsSkin aging TLR4 (+896 A/G
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